Dr. Fleming

	Department of Pathology
	Brigham and Women's Hospital
	A teaching Affliate of Harvard Medical School
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Name and Present Position:

MARK D. FLEMING, Associate Pathologist

Address:

Brigham and Women's Hospital, Department of Pathology, 75 Francis Street Boston, MA

Medical School (including school name, date and degree awarded):

1990 Dphil, University of Oxford, Oxford, United Kingdom
1993 MD, Harvard Medical School, Boston, MA

Graduate Medical Education (including dates and institution of internships, residencies, fellowships, etc):

1993-95 Resident, Anatomic Pathology, Brigham and Women's Hospital
1995-96 Fellow, Hematopathology, Brigham and Women's Hospital
1997-99 Research Fellow, Division of Hematology/Oncology, Children's Hospital, Boston, MA

Certification:

1998 Diplomate, American Board of Pathology (Anatomic Pathology)
1998 Diplomate, American Board of Pathology (Hematology)

Current Academic Appointment:

Assistant Professor of Pathology, Harvard Medical School

Current Hospital Appointments:

Associate Pathologist, Brigham and Women's Hospital
Staff Pathologist, Children's Hospital
Pathologist, Dana Farber Cancer Institute

Selected Bibliography:

Fleming MD, Trenor CC, Su M, Foernzler D, Beier D, Dietrich W, and Andrews NC. Microcytic anaemia mice have a
mutation in Nramp2, a candidate iron transporter gene. Nature Genetics 1997;16:383-386.
Fleming MD, Romano MA, Garrick L, Garrick MD and Andrews NC. The iron transporter Nramp2 is mutated in the
anemic Belgrade rat: Evidence of a role for Nramp2 in endosomal iron transport. PNAS1998, 95:1148-1153.
Fleming, MD. Hepatic iron overload in the age of hereditary hemochromatosis mutation analysis [editorial]. 1998 AJCP 109: 505-507.
Su, MA, Trenor, C, Fleming, JC, Fleming, MD, and Andrews, NC. The G185R mutation disrupts function of the iron transporter Nramp2. 1998 Blood 92:6, 2157-2163.
Fleming, MD and Andrews, NC. Mammalian iron transport: an unexpected link between metal homeostasis and host
defense. 1998 J. Lab. Clin. Med. 132:464-468.
Levy, JE, Montross, LK, Cohen, DE, Fleming, MD, and Andrews, NC. The C282Y mutation causing hereditary
hemochromatosis does not produce a null allele. 1999 Blood, 94 (1):9-11.